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Researchers at the ICN2 have resolved one of the great unknowns in bone self-repair: how the cells responsible for forming new bone tissue are called into action.
Their work reveals the role of an electromechanical phenomenon at the nanoscale, flexoelectricity, as a possible mechanism for stimulating the cell response and guiding it throughout the fracture repair process. It has potential implications for the prosthetics industry and the development of biomimetic self-healing materials. The findings were published in Advanced Materials. Following this research, a cytological study of the interaction between crack stress and bone cells was published in Scientific Reports. You will find the links to both articles below.
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