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Copper is a component of many body proteins; almost all of the body’s copper is bound to copper proteins. Unbound (free) copper ions are toxic. Genetic mechanisms control the incorporation of copper into apoproteins and the processes that prevent toxic accumulation of copper in the body. Acquired copper toxicity can result from ingesting or absorbing excess copper (eg, from ingesting an acidic food or beverage that has had prolonged contact with a copper container). Self-limited gastroenteritis with nausea, vomiting, and diarrhea may occur. Copper toxicity causes acute intravascular hemolytic anemia, icterus, and hemoglobinuric nephrosis.

Copper toxicity is a type of metal poisoning caused by an excess of copper in the body. Copperiedus can occur from eating acidic foods cooked in uncoated copper cookware, or from exposure to excess copper in drinking water, an IUD or other environmental sources. More severe toxicity results from ingestion (usually with suicidal intent) of gram quantities of a copper salt (eg, copper sulfate) or from absorption of large amounts through the skin (eg, if compresses saturated with a solution of a copper salt are applied to large areas of burned skin). Hemolytic anemia and anuria can result and may be fatal.

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